Question:
Which of the following is NOT a component of crush syndrome?
Which of the following is NOT a component of crush syndrome?
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Crush syndrome is built around muscle breakdown, myoglobin release and kidney injury, not a bleeding disorder.
Updated On: Jul 8, 2026
- Myoglobinuria
- Massive crushing of muscles
- Acute tubular necrosis
- Bleeding diathesis
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Verified By Collegedunia
The Correct Option is D
Solution and Explanation
Step 1: Understanding the Question:
Crush syndrome develops when a limb or a large muscle mass is trapped and crushed for a long time, as happens to people pinned under rubble or heavy machinery. We need to pick the finding that is NOT part of this syndrome.
Step 2: Key Formula or Approach:
Crush syndrome runs on three linked events: heavy muscle damage from the crush itself, release of muscle contents such as myoglobin, potassium and phosphate into the blood, and kidney injury caused by that myoglobin load. Check each option against this picture.
Step 3: Detailed Explanation:
Massive crushing of muscle is the injury that starts the whole process, so it is a true part of the syndrome, not the answer.
When muscle fibers break down, they release myoglobin into the blood. This myoglobin is filtered by the kidney and shows up in the urine as myoglobinuria, turning it dark and tea colored. This is a core, expected finding.
The filtered myoglobin is toxic to the kidney tubule cells, more so when the patient is dehydrated and acidotic, and this produces acute tubular necrosis. This is exactly why crush syndrome patients get aggressive fluids and urine alkalinisation early. It is also a core finding.
Bleeding diathesis means an abnormal tendency to bleed. This is not part of the classic picture of crush syndrome, which centers on muscle breakdown, myoglobin release, hyperkalemia, acidosis and kidney injury. A severe crush injury can rarely push a critically ill patient into disseminated intravascular coagulation, but that is a late complication of critical illness in general, not a defining feature of crush syndrome itself.
Step 4: Final Answer:
Bleeding diathesis is not a recognised component of crush syndrome, so option 4 is correct.
Crush syndrome develops when a limb or a large muscle mass is trapped and crushed for a long time, as happens to people pinned under rubble or heavy machinery. We need to pick the finding that is NOT part of this syndrome.
Step 2: Key Formula or Approach:
Crush syndrome runs on three linked events: heavy muscle damage from the crush itself, release of muscle contents such as myoglobin, potassium and phosphate into the blood, and kidney injury caused by that myoglobin load. Check each option against this picture.
Step 3: Detailed Explanation:
Massive crushing of muscle is the injury that starts the whole process, so it is a true part of the syndrome, not the answer.
When muscle fibers break down, they release myoglobin into the blood. This myoglobin is filtered by the kidney and shows up in the urine as myoglobinuria, turning it dark and tea colored. This is a core, expected finding.
The filtered myoglobin is toxic to the kidney tubule cells, more so when the patient is dehydrated and acidotic, and this produces acute tubular necrosis. This is exactly why crush syndrome patients get aggressive fluids and urine alkalinisation early. It is also a core finding.
Bleeding diathesis means an abnormal tendency to bleed. This is not part of the classic picture of crush syndrome, which centers on muscle breakdown, myoglobin release, hyperkalemia, acidosis and kidney injury. A severe crush injury can rarely push a critically ill patient into disseminated intravascular coagulation, but that is a late complication of critical illness in general, not a defining feature of crush syndrome itself.
Step 4: Final Answer:
Bleeding diathesis is not a recognised component of crush syndrome, so option 4 is correct.
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