Answer: Paralytic squint.
Step 1: The hallmark in the stem is secondary deviation greater than primary deviation. This is the classic sign of a paralytic (incomitant) squint.
Step 2: When the patient fixes with the paralysed eye, extra innervation flows to both eyes (Hering's law), so the normal eye over-deviates: secondary deviation exceeds primary deviation.
Step 3: The clinical setting fits. A 65-year-old diabetic and hypertensive is prone to microvascular cranial nerve palsies (III, IV, VI) causing acute diplopia and squint.
Why not the others: In concomitant squint the deviation is equal in all gaze positions (primary = secondary). Restrictive squint is due to mechanical tethering, not the diabetic nerve palsy picture. Pseudosquint is only an apparent deviation with normal alignment.
Ref: GPonline, Basics of Strabismus.